Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. On the flip side, our capacity to rescue SERT purpose in KI midbrain synaptoneurosomes because of the inhibition of FAK suggests elevated FAK signaling downstream in the Pro32Pro33 mutant, as confirmed by greater pFAK localization https://pro33login22221.bleepblogs.com/32801597/how-pro33-can-save-you-time-stress-and-money