Preliminary reports show conolidine may perhaps inhibit certain ion channels, minimizing neuronal excitability and limiting suffering signals. This system is especially related in neuropathic suffering, in which abnormal signaling brings about persistent distress. Moreover, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to ache perception. Computation... https://antoniog225jfa4.wikibyby.com/user